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Reducing Birth Defects With Higher Dose Folic Acid

These studies show a dose-dependent relationship, where 0.4 mg of folic acid reduced spina bifida by about 48% and 4.0 mg reduces it about 72%.

A potential problem with taking folic acid in doses as high as 4.0 mg is that folic acid can mask a B12 deficiency. Taking B12 when folic acid is taken can reduce this risk.

However, as with all things concerned with pregnancy, the information provided here should not be construed as personal medical advice. It is simply educational material.

Women who are pregnant should present information like this to their doctor when seeking medical advice, and work with their doctor in making decisions related to their pregnancy.

Prevention of neural tube defects with periconceptional folic acid.
Clin Perinatol 1994 Sep;21(3):483-503
Rieder MJ

A body of convincing evidence demonstrates that periconceptional folic acid supplements are associated with a reduction in the risk for first occurrences of neural tube defect and for recurrence of neural tube defects among women with a previously affected pregnancy. The mechanism(s) of this protective effect are unknown, and although the effect can be seen with folate doses between 0.4 and 4 mg, the optimal dose remains unknown. The optimal method of ensuring that all women of child-bearing age receive this dose of folate remains in question; supplementation offers the advantage of targeting only the population at risk, whereas food fortification offers the advantage of ensuring compliance in virtually all of the population at risk. The use of periconceptional folic acid can be expected to reduce, but not abolish, the incidence of neural tube defects. Research into the mechanism(s) of the protective effects of folic acid and the underlying biology of neural tube defects is required to address the problem of how to develop therapeutic strategies to further reduce the impact of neural tube defects.

Folate levels and neural tube defects. Implications for prevention [see comments]
JAMA 1995 Dec 6;274(21):1698-702
Daly LE; Kirke PN; Molloy A; Weir DG; Scott JM

Using data from a recent case-control study, a woman's risk of having a child with a neural tube defect (NTD) was found to be associated with early pregnancy red cell folate levels in a continuous dose-response relationship. These findings were used to calculate the reduction in NTD cases that would be expected under two different strategies to raise folate levels. Targeting high-risk individuals has a small effect on the population prevalence but can substantially change an individual's risk. Targeting the population produces a small change in individual risk but has a large effect on the population prevalence. Supplementation of high-risk women would be the most efficient method to implement the high-risk strategy, while food fortification would be preferable for the population approach. The current guidelines for the prevention of NTD are for an increased folic acid intake of 0.4 mg per day. This would result in a 48% reduction in NTDs, which may be near optimal. The two intervention strategies should be considered complementary in prevention of NTDs.

Recommendations on the use of folic acid supplementation to prevent the recurrence of neural tube defects. Clinical Teratology Committee, Canadian College of Medical Geneticists [see comments]
CMAJ 1993 Nov 1;149(9):1239-43 (ISSN: 0820-3946)
Van Allen MI; Fraser FC; Dallaire L; Allanson J; McLeod DR; Andermann E; Friedman JM

OBJECTIVE: To prevent the recurrence of neural tube defects (NTDs) in families at increased risk of having offspring with NTDs with the use of periconceptional folic acid supplementation. OPTIONS: Genetic counselling and prenatal diagnosis of NTDs. OUTCOMES: NTDs cause stillbirth, neonatal death and severe disabilities. The cost for medical care and rehabilitation in the first 10 years of life of a child with spina bifida cystica was estimated to be $42,507 in 1987. EVIDENCE: The authors reviewed the medical literature, communicated with investigators from key studies, reviewed policy recommendations from other organizations and drew on their own expertise. A recent multicentre randomized controlled trial showed that among women at high risk of having a child with an NTD those who received 4 mg/d of folic acid had 72% fewer cases of NTD-affected offspring than nonsupplemented women. Two previous intervention studies also demonstrated that folic acid supplementation was effective in reducing the rate of NTD recurrence. Several retrospective studies support this conclusion.

VALUES: Recommendations are the consensus of the Clinical Teratology Committee of the Canadian College of Medical Geneticists (CCMG) and have been approved by the CCMG Board. The committee believes that primary prevention of NTDs is preferable to treatment or to prenatal detection and abortion.

BENEFITS, HARMS AND COSTS: Folic acid supplementation should result in fewer NTDs among infants in Canada and ancillary savings in medical costs. The recommended dosage of folic acid is not known to be associated with adverse effects. Higher dosages of folic acid may make vitamin B12 deficiency difficult to diagnose and may alter seizure frequency in patients with epilepsy due to drug interactions with anticonvulsants.

RECOMMENDATIONS: A minimum dosage of folic acid of 0.8 mg/d, not to exceed 5.0 mg/d, is recommended along with a well-balanced, nutritious diet for all women who are at increased risk of having offspring with NTDs and who are planning a pregnancy or may become pregnant. Supplementation should begin before conception and continue for at least 10 to 12 weeks of pregnancy.

VALIDATION: These guidelines are similar to those of the Society of Obstetricians and Gynaecologists of Canada, the US Centers for Disease Control and Prevention and the Department of Health in Britain. SPONSORS: These guidelines were developed by the CCMG Clinical Teratology Committee and endorsed by the Board of the CCMG. No funding for the development of these guidelines was obtained from any other sources.

Folic acid and neural tube defects: the current evidence and implications for prevention.
Ciba Found Symp 1994;181:192-208; discussion 208-11 (ISSN: 0300-5208)
Wald NJ

The results of the MRC Vitamin Study have established the specific role of folic acid in the prevention of neural tube defects. Folic acid supplementation (4 mg/day) at the time of conception reduced the recurrence rate by about 70%. Evidence from observational studies and the Hungarian randomized trial (that used 0.4-0.8 mg/day supplemental folic acid) indicates that a similar level of prevention can be achieved among women who have not already had an affected pregnancy and that this may be achieved with a lower dose of folic acid. The medical implications of these results are important. (1) Women planning a pregnancy should take folic acid supplements. (2) Because a large proportion of pregnancies are unplanned and many of those that are planned will occur in women who will not have taken folic acid supplements, a complementary general public health strategy is needed, based on dietary advice and food fortification designed to achieve the benefit with adequate safety. (3) Any public health prevention strategy needs to be monitored for efficacy and possible harm. (4) Research is needed on the dose-response relationship between folic acid and neural tube defect prevention and the mechanism of action.

Peri-conception use of folic acid in the prevention of neural tube defect: current concepts
[Uso periconcepcional de acido folico en la prevencion de defectos del tubo neural: conceptos actuales.]
Ginecol Obstet Mex 1996 Sep;64:418-21 (ISSN: 0300-9041)
Garcia-Morales MA; Limon-Luque LM; Barron-Vallejo J; Gonzalez-Morales ME; Kably-Ambe A

The incidence of neural tube defects (NTDs) is about 1.3 cases per 1000 live births. The higher incidence of NTDs occurs among certain ethnic groups, and geographic areas. The fetal morbimortality is high and the treatment for those babies who live is expensive with pour quality of live. It is unclear what biochemical mechanism involving folate explain the relationship of this vitamin to the pathogenesis of NTDs. However elevated concentrations of homocysteine or decreased methionine concentrations could be interfere with closure of the neural tube. The pharmacologic periconceptional intake of 0.4-4.0 mg/day of folic acid reduces the risk of occurrent NTDs by approximately 40-75%. A relatively high dietary intake of folate may also reduce the risk.

Prospects for prevention of neural tube defects by vitamin supplementation.
Curr Opin Neurol Neurosurg 1993 Aug;6(4):554-8 (ISSN: 0951-7383)
Mills JL; Simpson JL

Recent studies have resolved the debate over the role of vitamins in preventing neural tube defects. The British Medical Research Council trial demonstrated that 4 mg of folate daily, but not other vitamins, prevented 72% of recurrences. The Hungarian trial prevented neural tube defects in women who had not previously had affected children by giving multivitamins containing 0.8 mg of folate. The US Public Health Service currently recommends that women at risk for becoming pregnant take 0.4 mg of folate daily. Unfortunately, most pregnancies are unplanned, and women not planning to become pregnant may not follow this recommendation. Therefore, the US Food and Drug Administration is exploring methods of food fortification. Because large doses of folate have been reported to ameliorate B12 deficiency anemia while allowing neurologic damage to progress, and to cause electroencephalogram abnormalities in epileptics, it is important to plan fortification carefully and to monitor both toxicity and benefits.

Folic Acid Reduces Homocysteine
Homocysteine induces congenital defects of the heart and neural tube: effect of folic acid.
Proc Natl Acad Sci U S A 1996 Dec 24;93(26):15227-32 (ISSN: 0027-8424)
Rosenquist TH; Ratashak SA; Selhub J

The biological basis or mechanism whereby folate supplementation protects against heart and neural tube defect is unknown. It has been hypothesized that the amino acid homocysteine may be the teratogenic agent, since serum homocysteine increases in folate depletion; however, this hypothesis has not been tested. In this study, avian embryos were treated directly with D,L-homocysteine or with L-homocysteine thiolactone, and a dose response was established. Of embryos treated with 50 microliters of the teratogenic dose (200 mM D,L-homocysteine or 100 mM L-homocysteine thiolactone) on incubation days 0, 1, and 2 and harvested at 53 h (stage 14), 27% showed neural tube defects. To determine the effect of the teratogenic dose on the process of heart septation, embryos were treated during incubation days 2, 3, and 4; then they were harvested at day 9 following the completion of septation. Of surviving embryos, 23% showed ventricular septal defects, and 11% showed neural tube defects. A high percentage of the day 9 embryos also showed a ventral closure defect. The teratogenic dose was shown to raise serum homocysteine to over 150 nmol/ml, compared with a normal level of about 10 nmol/ml. Folate supplementation kept the rise in serum homocysteine to approximately 45 nmol/ml, and prevented the teratogenic effect. These results support the hypothesis that homocysteine per se causes dysmorphogenesis of the heart and neural tube, as well as of the ventral wall.

Folic Acid for the Prevention of Neural Tube Defects (RE9834)
From: http://www.aap.org/policy/re9834.html
Web site of the American Academy of Pediatrics
American Academy of Pediatrics Committee on Genetics

ABSTRACT

The American Academy of Pediatrics endorses the US Public Health Service (USPHS) recommendation that all women capable of becoming pregnant consume 400 µg of folic acid daily to prevent neural tube defects (NTDs). Studies have demonstrated that periconceptional folic acid supplementation can prevent 50% or more of NTDs such as spina bifida and anencephaly. For women who have previously had an NTD-affected pregnancy, the Centers for Disease Control and Prevention (CDC) recommends increasing the intake of folic acid to 4000 µg per day beginning at least 1 month before conception and continuing through the first trimester. Implementation of these recommendations is essential for the primary prevention of these serious and disabling birth defects. Because fewer than 1 in 3 women consume the amount of folic acid recommended by the USPHS, the Academy notes that the prevention of NTDs depends on an urgent and effective campaign to close this prevention gap.

ABBREVIATIONS

NTDs, neural tube defects; USPHS, US Public Health Service; CDC, Centers for Disease Control and Prevention; MRC, Medical Research Council; IOM, Institute of Medicine; AAP, American Academy of Pediatrics.

BACKGROUND

Neural tube defects (NTDs) are among the most common birth defects contributing to infant mortality and serious disability. NTDs, which include anencephaly, spina bifida, and encephalocele, occur in approximately 1 of 1000 births in the United States.1 An estimated 4000 pregnancies are affected with NTDs each year. More than one third of these pregnancies are spontaneously lost or electively terminated; thus, about 2500 infants per year are born with an NTD. The results of 2 randomized controlled trials and several observational studies showed that 50% or more of NTDs can be prevented if women consume a folic acid-containing supplement before and during the early weeks of pregnancy2,3 in addition to the folate in their diet. Based on a synthesis of these data, the US Public Health Service (USPHS) and Centers for Disease Control and Prevention (CDC) recommendations were developed.4,5 Because the evidence for folic acid prevention evolved over time, there are two separate recommendations: one for women who have no history of a previous NTD-affected pregnancy and one for women who have had a previous NTD-affected pregnancy.

WOMEN WITH NO HISTORY OF A PREVIOUS NTD-AFFECTED PREGNANCY

Of children with an NTD, 95% are born to couples with no family history of these defects. Evidence to date suggests that supplementation with a multivitamin containing 400 (0.4 mg) µg of folic acid prevents the occurrence of >50% of NTDs when it is taken before conception and continued throughout the first trimester of pregnancy.5 The USPHS recommends that all women of childbearing age who are capable of becoming pregnant take 400 µg of folic acid daily.5 Implementing this recommendation may provide the opportunity for primary prevention of 50% or more of these serious disabling birth defects. Regular and ongoing ingestion of folic acid by women of childbearing age is necessary because approximately half of the pregnancies in the United States are unplanned,6 and neural tube closure occurs during the first 4 weeks of gestation.7 Despite the publication of the USPHS recommendation in September 1992, a 1998 poll showed that 70% of women aged 18 to 45 years still are not following the USPHS recommendation.8

WOMEN WHO HAVE HAD A PREVIOUS NTD-AFFECTED PREGNANCY

Among US couples who have had a child with an NTD, the recurrence risk is 2% to 3% in subsequent pregnancies.9 In 1991, the Medical Research Council (MRC) Vitamin Study Group reported the results of a well-designed, prospective, randomized trial of folic acid supplementation for the prevention of NTDs in pregnancies of women who had a previous child with an NTD, and the CDC published its recommendations for consumption of 4000 (4 mg) µg of folic acid.4 The results of the MRC study conclusively demonstrated that a daily dosage of 4000 µg of folic acid, in addition to folate in the diet, before and during early pregnancy resulted in a 71% reduction of recurrence of NTDs. The addition of other vitamins to the dosage of folic acid did not reduce the risk further. Use of multivitamins without folic acid did not result in a reduced risk for NTDs. The MRC study did not explore the possible benefit of a dosage lower than 4000 µg of folic acid. However, an earlier nonrandomized study conducted in the United Kingdom suggested that a lower dosage, 360 µg daily, resulted in a comparable reduction of recurrence of NTDs.10 Although adverse maternal or fetal effects of a daily 4000 µg dosage of folic acid were not detected by the MRC study, the small size of the study groups precluded detection of uncommon adverse effects.

FOLATE AND FOLIC ACID

Folic acid, also known as pteroylmonoglutamic acid, is a synthetic compound used in dietary supplements and fortified foods. The term folate includes all compounds that have the vitamin properties of folic acid--including folic acid and naturally occurring compounds in food.11 The average diet in the United States contains 200 µg of naturally occurring food folate, which is less bioavailable than folic acid.12 Additional intake of foods rich in folate could raise the average intake, but it has not been demonstrated that increased consumption of food folate would prevent NTDs as effectively as a daily vitamin supplement containing 400 µg of folic acid. A small comparison study suggests that blood folate concentrations are increased much more by folic acid supplementation than by naturally occurring food folate in the diet.13 Economic and social circumstances may make an adequate increase in dietary folate difficult or unlikely, and the behavioral change required among a large fraction of women may take years to achieve.

Folic acid is a water-soluble vitamin that has no known toxicity. However, higher doses of folic acid can correct the anemia of vitamin B12 deficiency (pernicious anemia), which might be an important clue to the presence of vitamin B12 deficiency in some instances. Folic acid does not prevent the neurologic consequences of vitamin B12 deficiency, and, for this reason, the USPHS recommendation cautioned that intake of folate should be not >1000 µg per day. However, the Institute of Medicine (IOM) Food and Nutrition Board recently set the tolerable upper intake limit of synthetic folic acid at 1000 µg, thus eliminating food folate from the calculation.14 Because pernicious anemia rarely occurs before the age of 50 years, it is likely to be rare among women consuming folic acid during the reproductive years. Folic acid has been consumed by about a quarter of all women for many years and extensively during later pregnancy without apparent adverse effects; however, studies that definitively address the question of maternal and fetal safety of folic acid are not available.

The IOM Food and Nutrition Board's recommended dietary allowance (RDA) for folate is 400 µg for adults and 600 µg for pregnant women.14 To reduce the risk for NTDs, the IOM recommended that women capable of becoming pregnant consume 400 µg of folic acid daily from fortified foods, vitamin supplements, or a combination of the two. This is in addition to the naturally occurring folate obtained from a varied diet.14 The majority of multivitamin preparations contain 400 µg of folic acid. These preparations are available over the counter and are already being taken by about 30% of nonpregnant women aged 18 to 45 years in the United States.8 Tablets containing folic acid alone are available over the counter in dosages up to 800 µg but the availability is very limited when compared with multivitamin preparations. Folic acid tablets in a 1000 µg dose are available by prescription only. This preparation is most frequently utilized by women who are taking 4000 µg because of a previous NTD-affected pregnancy.

In March 1996, the Food and Drug Administration mandated that enriched cereal-grain products be fortified with 140 µg of folic acid per 100 g of flour.15 This measure increases the proportion of women who consume the USPHS-recommended daily dosage of 400 µg of folic acid only an additional 3%, because this fortification level will provide the average woman only an additional 100 µg of folic acid per day (unpublished data, 1992).

RECOMMENDATIONS

1. Prevention for Women With No History of a Previous NTD-Affected Pregnancy. The American Academy of Pediatrics (AAP) endorses the USPHS recommendation that all women of childbearing age who are capable of becoming pregnant should consume 400 (0.4 mg) µg of folic acid daily. Because of the high rate of unplanned pregnancies in the United States, the AAP encourages efforts at devising a program of food fortification to provide all women a daily intake of 400 µg of folic acid. In the absence of optimal fortification, the AAP encourages women to consume 400 µg of folic acid daily in addition to eating a healthy diet. At present, the most convenient, inexpensive, and direct way to meet the recommended dosage is by taking a multivitamin containing 400 µg of folic acid, but efforts to increase the availability of folic acid-only supplements should be encouraged for women who prefer not to take multivitamins. Because the risk for NTDs is not totally eliminated by folic acid use, routine prenatal screening for NTDs is still advisable.

2. Prevention for Women Who Have Had a Previous NTD-Affected Pregnancy. Women with a history of a previous pregnancy resulting in a fetus with an NTD should be advised of the results of the MRC study. During times in which a pregnancy is not planned, these high-risk women should consume 4000 (4 mg) µg of folic acid per day. However, they should be offered treatment with 4000 µg of folic acid per day starting 1 month before the time they plan to become pregnant and throughout the first 3 months of pregnancy, unless contraindicated. Women should be advised not to attempt to achieve the 4000 µg daily dosage of folic acid by taking over-the-counter or prescription multivitamins containing folic acid because of the possibility of ingesting harmful levels of other vitamins, for example, Vitamin A.17 It should be noted that 4000 µg of folic acid did not prevent all NTDs in the MRC study. Therefore, high-risk patients should be cautioned that folic acid supplementation does not preclude the need for counseling or consideration of prenatal testing for NTDs.

3. Prevention for Other High-Risk Persons. No intervention or observational studies address prevention for other high-risk persons. Women with a close relative (eg, sibling, niece, or nephew) who has an NTD (risk is approximately 0.3% to 1.0%), women with type 1 diabetes mellitus (risk is approximately 1%), women with seizure disorders being treated with valproic acid or carbamazepine (risk is approximately 1%), and women or their partners who have an NTD (risk may be 2% to 3%)18 and are planning a pregnancy should discuss with their physician the risk for an affected child and the advantages and disadvantages of increasing their daily periconceptional folic acid intake to 4000 µg.

4. Public Health Programs: Supplementation, Surveillance, and Food Fortification. The AAP recommends that the Department of Health and Human Services expeditiously devise and implement an educational program to prevent folic acid-preventable NTDs throughout the use of supplements, fortified foods, or a combination of both. The program should support surveillance of effectiveness and adverse outcomes to further refine the effective folate dose and mechanisms of actions. In light of the recent IOM recommendation, the AAP also encourages additional efforts at devising a program of food fortification with folic acid to provide all women capable of becoming pregnant a daily intake of 400 µg of folic acid.

REFERENCES

1. Centers for Disease Control and Prevention. Surveillance for anencephaly and spina bifida and the impact of prenatal diagnosis. United States, 1985-1994. MMWR. 1996;44(SS-4):1-13
2. MRC Vitamin Study Research Group. Prevention of neural tube defects: results of the Medical Research Council Vitamin Study. Lancet. 1991;338:131-137
3. Czeizel AE, Dudás I. Prevention of the first occurrence of neural-tube defects by periconceptional vitamin supplementation. N Engl J Med. 1992;327:1832-1835
4. Centers for Disease Control and Prevention. Use of folic acid for prevention of spina bifida and other neural tube defects: 1983-1991. MMWR. 1991;40:513-516
5. Centers for Disease Control and Prevention. Recommendations for the use of folic acid to reduce the number of cases of spina bifida and other neural tube defects. MMWR. 1992;41:1-8
6. Forrest JD. Epidemiology of unintended pregnancy and contraceptive use. Am J Obstet Gynecol. 1994;170:1485-1489
7. Moore KL. Formulation of the trilaminar embryo. In: The Developing Human. Philadelphia, PA: WB Saunders Co; 1988:55-64
8. Centers for Disease Control and Prevention. Knowledge and use of folic acid by women of childbearing age--United States, 1995 and 1998. MMWR. 1999;48:325-327
9. Hall JG, Solehdin F. Genetics of neural tube defects. Ment Retard Dev Disabil. 1999;4:269-281
10. Smithells RW, Nevin NC, Seller MJ, et al. Further experience of vitamin supplementation for prevention of neural tube defect recurrences. Lancet. 1983;1:1027-1031
11. Cornel MC, Erickson JD. Comparison of national policies on periconceptional use of folic acid to prevent spina bifida and anencephaly. Teratology. 1997;55:134-137
12. Bailey LB. Folate requirements and dietary recommendations. In: Bailey LB, ed. Folate in Health and Disease. New York, NY: Marcel Dekker, Inc; 1995:123-151
13. Daly LE, Kirke PN, Molloy A, Weir DG, Scott JM. Folate levels and neural tube defects: implications for prevention. JAMA. 1995;274:1698-1702
14. Institute of Medicine. Dietary reference intakes: folate, other B vitamins, and choline. In: Dietary Reference Intakes for Thiamin, Riboflavin, Vitamin B6, Folate, Vitamin B12, Pantothenic Acid, Biotin, and Choline. Washington, DC: National Academy Press; 1998:(8)32
15. US Department of Health and Human Services, Food and Drug Administration. Food standards: amendment of the standards of identity for enriched grain products to require addition of folic acid. Federal Register. 1996;61:8781-8807
16. Oakley GP. Let's increase folic acid fortification and include vitamin B12. Am J Clin Nutr. 1997;65:1889-1890
17. Oakley GP Jr, Erickson JD. Vitamin A and birth defects: continuing caution is needed. N Engl J Med. 1995;333:1414-1415
18. Tolmie J. Neural tube defects and other congenital malformations of the central nervous system. In: Rimoin DL, Connor JM, Pyeritz RE, eds. Emery and Rimoin's Principles and Practice of Medical Genetics. New York, NY: Churchill Livingstone Inc; 1997:2145-2176